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Catherine Weisz, Ph.D.
Acting Chief, Section on Neuronal Circuitry
Dr. Weisz received a B.S. in neurobiology from Cornell University, an M.S. in biotechnology from Johns Hopkins University, and a Ph.D. in neuroscience from Johns Hopkins University School of Medicine. Her graduate work involved studies of synaptic inputs and electrical properties of cochlear type II spiral ganglion afferent neurons in the laboratories of Dr. Paul Fuchs and Dr. Elisabeth Glowatzki. Postdoctoral work in the laboratory of Dr. Karl Kandler at the University of Pittsburgh School of Medicine investigated the development of circuits between brainstem neurons involved in sound localization. In 2015, Dr. Weisz moved to the NIDCD where she became acting chief of the Section on Neuronal Circuitry. Dr. Weisz’s laboratory investigates the synaptic transmission and electrical properties of descending neuronal circuitry in the auditory brainstem and cochlea.
Neuronal Circuitry Section
- Weisz, C.J.C., Givens, R.S., Rubio, M.E., Kandler, K. Excitation by inter-axonal GABA spillover in a sound localization circuit. J. Neurosci, 2016 Jan 20; 36(3):911-25.
- Sturm JJ and Weisz CJC. Hyperactivity in the medial olivocochlear system is a common feature of tinnitus and hyperacusis in humans. J. Neurophys, Neuroforum 2015 Nov;114(5):2551–4.
- Clause A, Kim G, Weisz CJC, Rubsamen R, Vetter D, Kandler K. The precise temporal pattern of pre-hearing spontaneous activity is necessary for tonotopic map refinement. Neuron, 2014 May 21; 82(4), 822–835.
- Weisz CJC, Glowatzki E, Fuchs P. Excitability of type II cochlear afferents. J. Neurosci. 2014 Feb 5; 34(6), 2365–73.
- Weisz CJC, Lehar M, Hiel H, Glowatzki E, Fuchs PA. Synaptic transfer from outer hair cells to type II afferent fibers in the rat cochlea. J. Neurosci. 2012 Jul 11; 32(28):9528–36.
- Weisz C, Glowatzki E, Fuchs P. The postsynaptic Function of type II cochlear afferents. Nature 461, 2009 Oct 22; 1126–9.
- Weisz CJC, Raike RS, Soria-Jasso LE, Hess EJ. Potassium channel blockers inhibit the triggers of attacks in the calcium channel mouse mutant tottering. J. Neurosci, 2005 April; 25(16):4141–4145.